Author

C. Megino

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

X. Matias-Guiu· 2 shared A. Casanovas· 1 shared A. Olomi· 1 shared A. Perez· 1 shared A. Perramon-Guell· 1 shared A. Yeramian· 1 shared G. Altes· 1 shared J. Egea· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

X. Matias-Guiu

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Multiple endocrine defects in adult-onset Sprouty1/2/4 triple knockout mice

G. Altes, A. Olomi, A. Perramon-Guell, S. Hernandez, A. Casanovas, A. Perez, J. M. Diaz-Tocados, J. M. Valdivielso, C. Megino, R. Navaridas, X. Matias-Guiu, O. D. Klein, J. Egea, X. Dolcet, A. Yeramian, M. Encinas

This study investigates adult-onset Sprouty1/2/4 triple knockout mice, revealing endocrine defects without increased tumor incidence.

bioRxiv · Apr 18, 2024Physiology

ARID1A-deficient cells promote endometrial carcinoma progression and dissemination by reprograming tumor microenvironment.

C. Megino, X. Matias-Guiu, N. Eritja

This study shows ARID1A-deficient endometrial tumor cells reprogram the tumor microenvironment, promoting cancer progression and dissemination through chemokine secretion.

bioRxiv · Dec 14, 2022 · PreprintCancer Biology