Author

C. Murdoch

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. K. Banah· 2 shared L. Kang· 2 shared V. Musale· 2 shared A. C. Lay· 1 shared A. Hasib· 1 shared A. M. Dorward· 1 shared B. Dong· 1 shared B. Qi· 1 shared

Activity by Year

2023-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersPhysiology

A. K. Banah

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3 papers

Recent·Most cited

Knock out of the intracellular calcium conducting ion channel Mitsugumin 23 (MG23) protects against pressure overload induced left ventricular hypertrophy and cardiac dysfunction.

A. M. Dorward, G. B. Robertson, C. Sneddon, C. L. O'Rourke, I. H. Um, D. J. Harrison, M. Nishi, H. Takeshima, C. Murdoch, S. J. Pitt

This study shows that knocking out MG23 protects against pressure overload-induced cardiac hypertrophy and dysfunction by reducing Ca2+ leak from the sarcoplasmic reticulum.

bioRxiv · Jun 28, 2024 · PreprintPhysiology

A Novel Role of Hyaluronan and its Membrane Receptors, CD44 and RHAMM in Obesity-Related Glomerulopathy

B. Qi, V. Musale, X. Weng, A. K. Banah, C. Murdoch, A. C. Lay, K. J. Heesom, W. Ju, M. Bitzer, C. Hills, R. J. Coward, L. Kang

This study identifies HA and its receptors CD44/RHAMM as key drivers of obesity-related glomerulopathy, showing their role in renal inflammation, fibrosis, and dysfunction, and suggests they as therapeutic targets.

bioRxiv · Jun 22, 2024 · PreprintPhysiology

Extracellular Matrix Abnormalities Contribute to Cardiac Insulin Resistance and Associated Dysfunction in Diet-induced Obese Mice

V. Musale, C. Murdoch, A. K. Banah, A. Hasib, C. K. Hennayake, B. Dong, C. C. Lang, D. H. Wasserman, L. Kang

This study shows that ECM abnormalities contribute to cardiac insulin resistance and dysfunction in obese mice, with potential therapeutic implications.

bioRxiv · Nov 14, 2023Physiology