Author

C. Perrin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pharmacology & Toxicology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:clustering· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Borenstein· 1 shared A. Di Genova· 1 shared A. Gilg Soit Ilg· 1 shared A. Gonzalez-Perez· 1 shared A. Khandekar· 1 shared A. Poret· 1 shared A. Scherpereel· 1 shared A. Sexton-Oates· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperPharmacology & Toxicology

A. Borenstein

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Interference of Neuronal TrkB Signaling by the Cannabis-Derived Flavonoids Cannflavins A and B

J. Holborn, A. Walczyk Mooradally, C. Perrin, B. Alural, C. Aitchison, A. Borenstein, J. Khokhar, T. Akhtar, J. Lalonde

This study shows that cannflavins A and B inhibit TrkB signaling in neurons by preventing Arc protein accumulation and downstream signaling, without agonist activity on GPCRs.

bioRxiv · Feb 3, 2022Pharmacology & Toxicology

Cytolysin-positive Enterococcus faecalis is elevated in patients with chronic alcoholic pancreatitis

C. Perrin, V. Rebours, N. Trainel, C. S. Voican, G. Perlemuter, A.-M. Cassard, D. Ciocan

The paper finds that patients with chronic alcoholic pancreatitis have higher levels of cytolysin-positive Enterococcus faecalis compared to healthy controls and those with alcoholic hepatitis.

bioRxiv · Nov 30, 2021 · PreprintPhysiology

Disentangling heterogeneity of Malignant Pleural Mesothelioma through deep integrative omics analyses

L. Mangiante, N. Alcala, A. Di Genova, A. Sexton-Oates, A. Gonzalez-Perez, A. Khandekar, E. Bergstrom, J. Kim, C. Giacobi, N. Le-Stang, S. Boyault, C. Cuenin, S. Tabone-Eglinger, F. Damiola, C. Voegele, , Meyer

This study identifies four sources of heterogeneity in MPM using multi-omic factor analysis, showing how genomic events shape molecular profiles and clinical behavior.

bioRxiv · Sep 27, 2021Cancer Biology