Author

Chandni Parmar

University of Leicester

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 2 Organism:yeast· 2 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

Ábel VértesyiD· 2 shared Cyril DomingueziD· 2 shared Edda KlippiD· 2 shared Emma J. Kelsall· 2 shared Gabriele Schreiber· 2 shared Kayoko TanakaiD· 2 shared Kees R. StraatmaniD· 2 shared Mishal TariqiD· 2 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

Ábel VértesyORCID

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2 papers

Recent·Most cited

Constitutively active RAS prolongs Cdc42 signalling while attenuating MAPK signalling during fission yeast mating

Emma J. Kelsall, Akatsuki Kimura, Ábel Vértesy, Kees R. Straatman, Mishal Tariq, Raquel Gadea, Chandni Parmar, Gabriele Schreiber, Shubhchintan Randhawa, Takashi Y. Ida, Cyril Dominguez, Edda Klipp, Kayoko Tanaka

This study shows that constitutively active RAS in fission yeast prolongs Cdc42 signaling while reducing MAPK signaling through competitive binding and feedback mechanisms.

bioRxiv · Mar 6, 2025 · PreprintCell Biology

Constitutively active RAS in S. pombe causes persistent Cdc42 signalling but only transient MAPK activation

Emma J. Kelsall, Ábel Vértesy, Kees R. Straatman, Mishal Tariq, Raquel Gadea, Chandni Parmar, Gabriele Schreiber, Shubhchintan Randhawa, Cyril Dominguez, Edda Klipp, Kayoko Tanaka

The paper shows that constitutively active Ras1.G17V in S. pombe causes prolonged Cdc42 activation but only transient MAPKSpk1 activation, with a model explaining the dynamics.

bioRxiv · Jan 29, 2019 · PreprintMolecular Biology