Author

CITIID-NIHR BioResource COVID-19 Collaboration

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Assay:proteomics· 1

Frequent Co-Authors

B. Meng· 2 shared J. Bradley· 2 shared J. Choi· 2 shared A. Abdullahi· 1 shared A. Saito· 1 shared D. Collier· 1 shared D. Mallery· 1 shared D. Yamasoba· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersMicrobiology

B. Meng

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2 papers

Recent·Most cited

MicrobiologybioRxivMay 7, 2022

SARS-CoV-2 Spike N-Terminal Domain modulates TMPRSS2-dependent viral entry and fusogenicity

B. Meng, R. Datir, J. Choi…·R. K. Gupta

This study examines how mutations in the SARS-CoV-2 Spike N-Terminal Domain affect viral entry and fusogenicity, showing context-dependent modulation of spike function.

DOI 10.1016/j.celrep.2022.111220

humanproteomics

𝕏 PostBluesky

MicrobiologybioRxivDec 17, 2021

SARS-CoV-2 Omicron spike mediated immune escape, infectivity and cell-cell fusion

B. Meng, I. Ferreira, A. Abdullahi…·S. Zepeda

This study investigates the immune escape, infectivity, and cell-cell fusion properties of SARS-CoV-2 Omicron BA.1 compared to Delta, showing higher ACE2 affinity and antibody evasion but similar antiviral drug efficacy.

DOI 10.1038/s41586-022-04474-x

humanbulk RNA-seq

𝕏 PostBluesky