Author

D. A. Stanton

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

E. E. Pistilli· 3 shared H. E. Wilson· 3 shared A. M. Infante· 1 shared C. Montgomery· 1 shared E. N. Pugacheva· 1 shared H. Hazard-Jenkins· 1 shared J. N. Link· 1 shared M. G. Chapa· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCancer Biology

E. E. Pistilli

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pioglitazone administration restores a PPARG-dependent transcriptional network and ATP levels within skeletal muscles of mice implanted with patient-derived breast tumors

D. A. Stanton, H. E. Wilson, M. G. Chapa, J. N. Link, W. J. Geldenhuys, E. E. Pistilli

This study shows that pioglitazone restores PPARG-dependent gene expression and ATP levels in skeletal muscles of mice with breast cancer xenografts, improving fatigue resistance.

bioRxiv · Oct 4, 2021Cancer Biology

Breast cancer-associated skeletal muscle mitochondrial dysfunction and lipid accumulation is reversed by PPARG

H. E. Wilson, D. A. Stanton, E. E. Pistilli

This study shows BC-derived factors impair skeletal muscle mitochondrial function and lipid metabolism, which can be reversed by PPARG activation.

American Journal of Physiology-Cell Physiology · Apr 1, 2021Peer-reviewedCancer Biology

Skeletal muscle reprogramming by breast cancer regardless of treatment history or tumor molecular subtype

H. E. Wilson, D. A. Stanton, C. Montgomery, A. M. Infante, M. Taylor, H. Hazard-Jenkins, E. N. Pugacheva, E. E. Pistilli

This study identifies molecular changes in skeletal muscle caused by breast cancer, showing mitochondrial dysfunction across subtypes and no effect of treatment on gene expression.

bioRxiv · Oct 21, 2019Cancer Biology