Author

D. Abankwa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 1

Frequent Co-Authors

A. K. Najumudeen· 1 shared E. Peuhu· 1 shared E. Wang· 1 shared G. b. Manoharan· 1 shared G. Jacquemet· 1 shared H. Hamidi· 1 shared H. Parkkola· 1 shared I. L. Barsukov· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCancer Biology

A. K. Najumudeen

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Targeting a broad spectrum of KRAS-mutant cancers by hyperactivation-induced cell death

J. Lilja, J. Kaivola, J. R. W. Conway, J. Vuorio, H. Parkkola, P. Roivas, T. Varila, G. Jacquemet, E. Peuhu, E. Wang, U. Pentikäinen, I. M. D. Posada, H. Hamidi, A. K. Najumudeen, O. J. Sansom, , J. Ivaska

This study identifies SHANK3 as a RAS interactor that limits oncogenic KRAS signaling, and shows that disrupting its interaction with KRAS induces cell death in KRAS-mutant cancers.

bioRxiv · Sep 21, 2022 · PreprintCancer Biology

Potential of phenothiazines to synergistically block calmodulin and reactivate PP2A in cancer cells

S. Okutachi, G. b. Manoharan, D. Abankwa

The paper investigates phenothiazines' ability to synergistically inhibit calmodulin and activate PP2A, showing anti-cancer effects in cells with KRAS or BRAF mutations.

bioRxiv · Jun 16, 2021 · PreprintBiochemistry