Author

D. Blackwell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

A. E. Munsterberg· 1 shared C. Utting· 1 shared E. Poschl· 1 shared G. F. Mok· 1 shared I. C. Macaulay· 1 shared L. Folkes· 1 shared L. Mincarelli· 1 shared S. Sebastian· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. E. Munsterberg

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1 paper

Recent·Most cited

Muscle stem cell function is impaired in absence of Talpid3 - a gene required for primary cilia formation

V. Martinez-Heredia, D. Blackwell, S. Sebastian, T. Pearson, G. F. Mok, L. Mincarelli, C. Utting, L. Folkes, E. Poschl, I. C. Macaulay, U. C. Mayer, A. E. Munsterberg

This study shows that Talpid3 is essential for muscle stem cell self-renewal and regeneration, and pharmacological Wnt activation can restore regeneration in its absence.

bioRxiv · Oct 14, 2022Cell Biology