Author

D. Latour

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:biophysical modeling· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

H. Khedmatgozar· 2 shared M. Tripathi· 2 shared S. Dutta· 2 shared S. Nandana· 2 shared A. Haynes· 1 shared G. K. Patel· 1 shared I. Warraich· 1 shared L. Brandi· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersCell Biology

H. Khedmatgozar

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2 papers

Recent·Most cited

TIAM1 signaling drives prostatic budding and branching phenotypes and is a potential therapeutic target for BPH

H. Khedmatgozar, S. Dutta, M. Dominguez, D. Latour, M. Johnson, M. Fokar, I. Warraich, W. DeRiese, A. Haynes, R. J. Matusik, L. Brandi, S. Nandana, M. Tripathi

This study identifies TIAM1-RAC1 signaling as a driver of prostatic budding and branching in BPH and suggests it as a therapeutic target.

bioRxiv · Feb 2, 2024 · PreprintCell Biology

TBX2 driven switch from Androgen Receptor to Glucocorticoid Receptor signaling confers therapeutic resistance in Prostate Cancer

S. Dutta, G. K. Patel, H. Khedmatgozar, D. Latour, M. Tripathi, S. Nandana

This study identifies TBX2 as a molecular switch that represses AR and activates GR signaling, driving enzalutamide resistance in CRPC, and suggests SP2509 as a potential therapeutic strategy.

bioRxiv · May 7, 2023Cancer Biology