Author

R. J. Matusik

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:biophysical modeling· 1 Research Focus:differential expression· 1 Assay:protein structure· 1

Frequent Co-Authors

M. M. Grabowska· 2 shared T. C. Case· 2 shared W. N. Awadallah· 2 shared A. Haynes· 1 shared C. M. Vezina· 1 shared D. Latour· 1 shared E. Ricke· 1 shared H. Khedmatgozar· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCancer Biology

M. M. Grabowska

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Author profile →

3 papers

Recent·Most cited

TIAM1 signaling drives prostatic budding and branching phenotypes and is a potential therapeutic target for BPH

H. Khedmatgozar, S. Dutta, M. Dominguez, D. Latour, M. Johnson, M. Fokar, I. Warraich, W. DeRiese, A. Haynes, R. J. Matusik, L. Brandi, S. Nandana, M. Tripathi

This study identifies TIAM1-RAC1 signaling as a driver of prostatic budding and branching in BPH and suggests it as a therapeutic target.

bioRxiv · Feb 2, 2024 · PreprintCell Biology

Identification of genes required for enzalutamide resistance in castration-resistant prostate cancer cells in vitro

S. E. Kohrt, W. N. Awadallah, R. A. Phillips, T. C. Case, R. Jin, J. S. Nanda, X. Yu, P. E. Clark, Y. Yi, R. J. Matusik, P. D. Anderson, M. M. Grabowska

This study identifies three genes (ACAT1, MAP3K11, and PSMD12) that support enzalutamide resistance in castration-resistant prostate cancer cells in vitro.

bioRxiv · Mar 27, 2020Cancer Biology

Prostatic osteopontin expression is associated with symptomatic benign prostatic hyperplasia

P. Popovics, W. N. Awadallah, S. Kohrt, T. C. Case, N. L. Miller, E. Ricke, W. Huang, M. Ramirez-Solano, Q. Liu, C. M. Vezina, R. J. Matusik, W. A. Ricke, M. M. Grabowska

This study shows that osteopontin (OPN) is more abundant in symptomatic benign prostatic hyperplasia (S-BPH) compared to incidental BPH, and OPN secretion is stimulated by pro-inflammatory cytokines, driving pro-inflammatory gene expression in stromal cells.

bioRxiv · Dec 23, 2019Pathology