Author

D. M. Calcagno

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:scRNA-seq· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Toomu· 2 shared K. Huang· 2 shared K. R. King· 2 shared N. Taghdiri· 2 shared R. Weissleder· 2 shared A. Aguirre· 1 shared A. Chu· 1 shared A. E. Feldstein· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

A. Toomu

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Single-Cell Transcriptomic Analysis of Livers During NLRP3 Inflammasome Activation Reveals a Novel Immune Niche

D. M. Calcagno, A. Chu, S. Gaul, N. Taghdiri, A. Toomu, A. Leszczynska, B. Kaufmann, A. Wree, L. Geisler, H. M. Hoffman, A. E. Feldstein, K. R. King

This study uses single-cell transcriptomics to show NLRP3 activation in mice causes chronic extramedullary myelopoiesis and fibrosis in the liver.

bioRxiv · Mar 31, 2021 · PreprintImmunology

Csf1r-GCaMP5 Reporter Mice Reveal Immune Cell Communication in Vitro and in Vivo

N. Taghdiri, D. M. Calcagno, Z. R. Fu, K. Huang, R. H. Kohler, R. Weissleder, T. P. Coleman, K. R. King

The paper develops a calcium reporter mouse model to non-destructively study spatiotemporal calcium dynamics in immune cells, revealing intercellular communication in vitro and in vivo.

bioRxiv · Feb 24, 2021 · PreprintImmunology

Type I interferon responses to ischemic injury begin in the bone marrow of mice and humans and depend on Tet2, Nrf2, and Irf3

D. M. Calcagno, R. Ng, A. Toomu, C. Zhang, K. Huang, A. Aguirre, R. Weissleder, L. Daniels, Z. Fu, K. King

The paper shows type I interferon responses to ischemic injury start in the bone marrow of mice and humans, involving Tet2, Nrf2, and Irf3, and identifies ISG scores as a clinical biomarker.

bioRxiv · Jan 3, 2020 · PreprintImmunology