Author

D. M. Frausto

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Immunology· 1

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Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Zloza· 1 shared E. F. Giurini· 1 shared J. Reiser· 1 shared J. Zayas· 1 shared M. F. Mohamed· 1 shared R. Estupinian· 1 shared R. Roy· 1 shared S. H. Shafikhani· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

A. Zloza

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1 paper

Recent·Most cited

Overriding defective FPR chemotaxis signaling in diabetic neutrophil stimulates infection control in diabetic wound

R. Roy, J. Zayas, S. J. Wood, M. F. Mohamed, D. M. Frausto, R. Estupinian, E. F. Giurini, T. M. Kuzel, A. Zloza, J. Reiser, S. H. Shafikhani

The paper identifies impaired FPR1 chemotaxis signaling in diabetic neutrophils as a cause of infection vulnerability and shows that CCL3 can override this defect to improve infection control and healing.

bioRxiv · Sep 9, 2021Immunology