Author

E. F. Giurini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Zloza· 2 shared A. Morin· 1 shared D. M. Frausto· 1 shared J. Reiser· 1 shared J. Zayas· 1 shared K. H. Gupta· 1 shared M. F. Mohamed· 1 shared M. Williams· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

A. Zloza

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2 papers

Recent·Most cited

Inactivated SARS-CoV-2 Reprograms the Tumor Immune Microenvironment and Improves Murine Cancer Outcomes

E. F. Giurini, M. Williams, A. Morin, A. Zloza, K. H. Gupta

This study shows that inactivated SARS-CoV-2 converts immunologically cold tumors to hot by generating CD8+ T cells, improving cancer outcomes in melanoma and triple-negative breast cancer.

bioRxiv · Jun 30, 2022 · PreprintCancer Biology

Overriding defective FPR chemotaxis signaling in diabetic neutrophil stimulates infection control in diabetic wound

R. Roy, J. Zayas, S. J. Wood, M. F. Mohamed, D. M. Frausto, R. Estupinian, E. F. Giurini, T. M. Kuzel, A. Zloza, J. Reiser, S. H. Shafikhani

The paper identifies impaired FPR1 chemotaxis signaling in diabetic neutrophils as a cause of infection vulnerability and shows that CCL3 can override this defect to improve infection control and healing.

bioRxiv · Sep 9, 2021Immunology