J. Zayas

Overriding defective FPR chemotaxis signaling in diabetic neutrophil stimulates infection control in diabetic wound

The paper identifies impaired FPR1 chemotaxis signaling in diabetic neutrophils as a cause of infection vulnerability and shows that CCL3 can override this defect to improve infection control and healing.

Tumor protein D52 (TPD52) affects cancer cell metabolism by negatively regulating AMPK.

This study shows TPD52 inhibits AMPK activity, leading to metabolic defects in mice, highlighting its role in cancer metabolism.