Author

D. M. Vermilyea

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

D. A. Hogan· 2 shared A. H. Gifford· 1 shared A. W. Crocker· 1 shared C. J. Geiger· 1 shared G. A. O'Toole· 1 shared G. Wong· 1 shared J. de Anda· 1 shared K. A. Lewis· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

9 top collaborators · 24 mutual co-author links

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2 shared papersMicrobiology

D. A. Hogan

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Author profile →

2 papers

Recent·Most cited

Non-motile subpopulations of Pseudomonas aeruginosa repress flagellar motility in motile cells through a type IV pili- and Pel-dependent mechanism

K. A. Lewis, D. M. Vermilyea, S. S. Webster, C. J. Geiger, J. de Anda, G. Wong, G. A. O'Toole, D. A. Hogan

The paper shows non-motile Pseudomonas aeruginosa cells repress swarming motility in motile cells via type IV pili and Pel exopolysaccharide.

bioRxiv · Oct 21, 2021 · PreprintMicrobiology

Calprotectin-mediated zinc chelation inhibits Pseudomonas aeruginosa protease activity in cystic fibrosis sputum

D. M. Vermilyea, A. W. Crocker, A. H. Gifford, D. A. Hogan

This study shows that calprotectin chelates zinc to inhibit Pseudomonas aeruginosa protease activity in cystic fibrosis sputum, revealing a mechanism for zinc-dependent virulence regulation.

bioRxiv · Feb 25, 2021Microbiology