Author

D. Opulente

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Evolutionary Biology· 3 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:yeast· 4 Research Focus:chromatin & epigenetic regulation· 3 Approach:statistical model· 3 Assay:whole-genome sequencing· 3 Research Focus:ecology & biodiversity· 2 Research Focus:gene regulatory network inference· 2 Assay:DNA methylation· 1 Approach:experimental assay· 1

Frequent Co-Authors

C. T. Hittinger· 4 shared A. L. LaBella· 3 shared A. Rokas· 3 shared J. L. Steenwyk· 3 shared A. B. Hulfachor· 1 shared B. F. Eichman· 1 shared C. P. Kurtzman· 1 shared D. Libkind· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersEvolutionary Biology

C. T. Hittinger

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4 papers

Recent·Most cited

Signatures of optimal codon usage predict metabolic ecology in budding yeasts

A. L. LaBella, D. Opulente, J. L. Steenwyk, C. T. Hittinger, A. Rokas

This study shows that optimal codon usage in metabolic pathways correlates with ecological niches in budding yeasts, suggesting codon optimization can reveal metabolic ecology.

bioRxiv · Jul 22, 2020Evolutionary Biology

Variation and selection on codon usage bias across an entire subphylum

A. L. LaBella, D. Opulente, J. L. Steenwyk, C. T. Hittinger, A. Rokas

This study examines codon usage bias across 327 budding yeast species, finding that while genetic drift drives global patterns, translational selection influences many genes and codons.

bioRxiv · Apr 13, 2019Evolutionary Biology

Extensive loss of cell cycle and DNA repair genes in an ancient lineage of bipolar budding yeasts

J. L. Steenwyk, D. Opulente, J. Kominek, X.-X. Shen, X. Zhou, A. L. LaBella, N. P. Bradley, B. F. Eichman, N. Cadez, D. Libkind, J. DeVirgilio, A. B. Hulfachor, C. P. Kurtzman, C. T. Hittinger, A. Rokas

This study identifies two lineages in Hanseniaspora yeasts, showing the FEL lineage lost more cell cycle and DNA repair genes than the SEL lineage, likely due to accelerated evolution and increased mutational load.

bioRxiv · Feb 11, 2019Evolutionary Biology

Pathogenic Budding Yeasts Isolated outside of Clinical Settings

D. Opulente, Q. Langdon, K. Buh, M. Haase, K. Sylvester, R. V. Moriarty, M. Jarzyna, S. Considine, R. Schneider, C. T. Hittinger

The paper isolates pathogenic budding yeasts from non-clinical environments, finding associations with fruits, soil, and plants, challenging the assumption that these yeasts are only contaminants.

bioRxiv · Dec 4, 2018Microbiology