Author

D. Verbovetskiy

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:mouse· 5 Assay:proteomics· 5 Research Focus:cancer & tumor microenvironment· 3 Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2

Frequent Co-Authors

I. Krukovets· 5 shared O. Stenina-Adognravi· 5 shared S. Muppala· 4 shared J. Gajeton· 3 shared E. F. Plow· 2 shared M. T. Rahman· 2 shared A. Fleischman· 1 shared A. Vasanji· 1 shared

Activity by Year

2019-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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5 shared papersCancer Biology

I. Krukovets

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5 papers

Recent·Most cited

miR-467 Prevents Inflammation And Insulin Resistance In Response To Hyperglycemia

J. Gajeton, I. Krukovets, R. Yendamuri, D. Verbovetskiy, A. Vasanji, L. Sul, O. Stenina-Adognravi

This study shows miR-467a-5p reduces inflammation and insulin resistance by inhibiting TSP-1 under hyperglycemia, with effects lost in Western diet-fed mice.

bioRxiv · Dec 14, 2020Physiology

HYPERGLYCEMIA-INDUCED MIR-467 DRIVES TUMOR INFLAMMATION AND GROWTH IN BREAST CANCER

J. Gajeton, I. Krukovets, S. Muppala, D. Verbovetskiy, J. Zhang, O. Stenina-Adognravi

This study identifies miR-467 as a hyperglycemia-induced miRNA that promotes breast cancer growth by enhancing angiogenesis and macrophage infiltration.

bioRxiv · Jul 1, 2020Cancer Biology

Thrombospondin-4 mediates hyperglycemia- and TGF-beta-induced inflammation in breast cancer

S. Muppala, R. Xiao, J. Gajeton, D. Verbovetskiy, I. Krukovets, O. Stenina-Adognravi

This study shows that thrombospondin-4 promotes inflammation and macrophage infiltration in breast cancer under hyperglycemia and TGF-beta conditions.

bioRxiv · Jan 3, 2020Cancer Biology

The P387 Thrombospondin-4 Variant Promotes Accumulation of Macrophages in Atherosclerotic Lesions

S. Muppala, M. T. Rahman, I. Krukovets, D. Verbovetskiy, E. Pluskota, A. Fleischman, D. G. Vince, E. F. Plow, O. Stenina-Adognravi

This study investigates the role of the P387 TSP4 variant in promoting macrophage accumulation in atherosclerotic lesions, finding it enhances macrophage adhesion and inflammation in both mice and humans.

bioRxiv · Jun 11, 2019Pathology

Effect of Thrombospondin-4 on Pro-inflammatory Phenotype Differentiation and Apoptosis in Macrophages.

M. T. Rahman, S. Muppala, J. Wu, I. Krukovets, D. Solovjev, D. Verbovetskiy, E. F. Plow, O. Stenina-Adognravi

This study shows that TSP4 promotes pro-inflammatory macrophage differentiation and apoptosis, with P387-TSP4 being more active in these processes.

bioRxiv · May 9, 2019Cell Biology