Author

Dennis P. McDaniel

Uniformed Services University of the Health Sciences

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:mouse· 3 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

Caroline A. Kopsidas· 2 shared Clara Christine LoweiD· 2 shared Xiaoming ZhouiD· 2 shared Yuanyi FengiD· 2 shared Alexandru V. Korotcov· 1 shared Alison A. ChomiakiD· 1 shared Andrew K. KnutseniD· 1 shared Bernard J. DardzinskiiD· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersNeuroscience

Caroline A. Kopsidas

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Sustained Generation of Neurons Destined for Neocortex with Oxidative Metabolic Upregulation upon Filamin Abrogation

Caroline A. Kopsidas, Clara Christine Lowe, Dennis P. McDaniel, Xiaoming Zhou, Yuanyi Feng

The paper shows that abrogating filamin in mouse models of periventricular heterotopia increases neocortical neuron generation in the V-SVZ through oxidative metabolism and vascularization.

iScience · Jun 7, 2024Peer-reviewedNeuroscience

Nde1 is Required for Heterochromatin Compaction and Stability in Neocortical Neurons

Alison A. Chomiak, Yan Guo, Caroline A. Kopsidas, Dennis P. McDaniel, Clara Christine Lowe, Hongna Pan, Xiaoming Zhou, Qiong Zhou, Martin L. Doughty, Yuanyi Feng

This study identifies NDE1's role in heterochromatin compaction and stability in neocortical neurons, linking its dysfunction to neurodevelopmental and neuropsychiatric disorders.

iScience · May 5, 2022Peer-reviewedGenetics

Genetic inactivation of SARM1 axon degeneration pathway improves outcome trajectory after experimental traumatic brain injury based on pathological, radiological, and functional measures

Donald V. Bradshaw, Andrew K. Knutsen, Alexandru V. Korotcov, Genevieve M. Sullivan, Kryslaine L. Radomski, Bernard J. Dardzinski, Xiaomei Zi, Dennis P. McDaniel, Regina C. Armstrong

This study shows that genetic inactivation of SARM1 reduces axonal damage and improves functional outcomes after traumatic brain injury.

Acta Neuropathologica Communications · May 17, 2021Peer-reviewedNeuroscience