Author

Ding Jin

Yangzhou University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Organism:mouse· 1 Organism:plant (other)· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

Chenglin ChiiD· 2 shared Jianzhong ZhuiD· 2 shared Nanhua CheniD· 2 shared Sen JiangiD· 2 shared Wanglong ZhengiD· 2 shared Ziyan Sun· 2 shared Chaoyang Chen· 1 shared Hanrong Lv· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersMicrobiology

Chenglin ChiORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

G4 Eurasian avian-like H1N1 swine influenza viruses exhibit enhanced pathogenicity potential in mice and pigs

Jun Jiao, Ding Jin, Ziyan Sun, Chenglin Chi, Sen Jiang, Nanhua Chen, Wanglong Zheng, Chaoyang Chen, Weiwei Su, Xiaoyan Ding, Jianzhong Zhu

This study isolates and analyzes two G4 Eurasian avian-like H1N1 swine influenza viruses, showing they have enhanced pathogenicity in mice and pigs due to molecular features like HA receptor affinity and polymerase activity.

bioRxiv · May 12, 2026 · PreprintMicrobiology

ASFV early protein p30 suppresses antiviral type I IFN induction by targeting TRIM21 and RIG-I like receptor signaling adaptor MAVS

J B Zhang, Hanrong Lv, Ding Jin, Ziyan Sun, Chenglin Chi, Sizhuang Liu, Sen Jiang, Nanhua Chen, Wanglong Zheng, Jianzhong Zhu

This study identifies ASFV p30's role in suppressing antiviral type I IFN induction by interacting with TRIM21 and MAVS, disrupting TRIM21-mediated K27 polyubiquitination of MAVS.

Veterinary Microbiology · May 9, 2026Peer-reviewedImmunology