Author

E. De Braekeleer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

B. J. P. Huntly· 1 shared G. S. Vassiliou· 1 shared J. S. L. Yu· 1 shared K. Tzelepis· 1 shared K. Yusa· 1 shared M. Gozdecka· 1 shared M. Gu· 1 shared S. H. Ong· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

B. J. P. Huntly

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1 paper

Recent·Most cited

KAT7 is a therapeutic vulnerability of MLL-rearranged acute myeloid leukemia

Y. Z. Au, M. Gu, E. De Braekeleer, J. S. L. Yu, S. H. Ong, M. Gozdecka, X. Chen, K. Tzelepis, B. J. P. Huntly, G. S. Vassiliou, K. Yusa

This study identifies KAT7 as a critical therapeutic target in MLL-rearranged AML by showing its role in maintaining acetylation marks and supporting cell proliferation.

bioRxiv · Apr 25, 2020 · PreprintCancer Biology