Author

E. Diaz-Beneitez

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Organism:chicken / avian· 1 Organism:human· 1 Organism:virus· 1

Frequent Co-Authors

D. Fuentes· 2 shared D. Rodriguez· 2 shared J. F. Rodriguez· 2 shared L. Broto· 2 shared C. Courtillon· 1 shared D. S. Ferrero· 1 shared F. Mendez· 1 shared I. Busnadiego· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersMicrobiology

D. Fuentes

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Author profile →

2 papers

Recent·Most cited

Type I Interferon acts as a major barrier to the establishment of infectious bursal disease virus (IBDV) persistent infections

L. Broto, N. Romero, F. Mendez, E. Diaz-Beneitez, O. Candelas-Rivera, D. Fuentes, L. Cubas-Gaona, C. Courtillon, N. Eterradosi, S. Soubies, J. R. Rodriguez, D. Rodriguez, J. F. Rodriguez

This paper shows that type I interferon inactivation enables IBDV to establish persistent infections in avian and human cells by reducing apoptosis.

bioRxiv · Oct 9, 2020Microbiology

Modelling the interaction between Infectious Bursal Disease Virus VP3 polypeptide and dsRNA: identification of key residues for ribonucleoprotein assembly and virus replication

I. Busnadiego, M. T. Martin, D. S. Ferrero, M. G. Millan de la Blanca, L. Broto, E. Diaz-Beneitez, D. Fuentes, D. Rodriguez, N. Verdaguer, L. Kremer, J. F. Rodriguez

This study identifies key residues in IBDV VP3 for dsRNA binding and virus replication by analyzing mutant versions and their effects on ribonucleoprotein assembly and host evasion.

bioRxiv · Aug 6, 2020 · PreprintMicrobiology