Author

E. Dubois-Deruy

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:rat· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

L. Bertrand· 2 shared A. Loyens· 1 shared C. Beauloye· 1 shared C. Dessy· 1 shared F. Pinet· 1 shared H. Esfahani· 1 shared J.-L. Balligand· 1 shared L. Bultot· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCell Biology

L. Bertrand

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Author profile →

2 papers

Recent·Most cited

Restore mitophagy is essential to prevent cardiac oxidative stress during hypertrophy

V. Peugnet, M. Chwastyniak, S. Lancel, L. Bultot, N. Fourny, O. Beseme, A. Loyens, W. Heyse, P. Amouyel, L. Bertrand, F. Pinet, E. Dubois-Deruy

This study investigates the role of mitochondrial oxidative stress in cardiac hypertrophy and evaluates the effects of two antioxidants, MitoQ and EUK 134, on cardiomyocyte function and hypertrophy.

bioRxiv · Jan 12, 2021 · PreprintCell Biology

Beta-3 Adrenoreceptors protect from hypertrophic remodelling through AMP-Activated Protein Kinase and Autophagy.

E. Dubois-Deruy, R. Gelinas, C. Beauloye, H. Esfahani, C. Dessy, L. Bertrand, J.-L. Balligand

This study shows that beta3-adrenergic receptors reduce cardiac hypertrophy by activating AMPK and preserving autophagy in both in vitro and in vivo models.

bioRxiv · Jan 14, 2020Cell Biology