Author

H. Esfahani

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Physiology· 3 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:mouse· 4 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. Dessy· 3 shared J.-L. Balligand· 3 shared C. Beauloye· 2 shared C. Bouzin· 2 shared L. Bertrand· 2 shared A. Marino· 1 shared B. Bouchard· 1 shared C. Des Rosiers· 1 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersPhysiology

C. Dessy

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

When the liver is in poor condition, so is the heart - cardiac remodelling in MASH mouse models

S. Bott, J. Lallement, A. Marino, E. P. Daskalopoulos, C. Beauloye, H. Esfahani, C. Dessy, I. Leclercq

This study examines cardiac remodelling in MASH mouse models, finding adverse structural changes in the heart linked to liver disease, not overfeeding or aging.

bioRxiv · May 1, 2024Physiology

A matter of food and substrain: obesogenic diets induce differential severity of cardiac remodeling and diastolic dysfunction in C57Bl/6J and C57Bl/6N substrains

L. Cascarano, H. Esfahani, P. Michel, C. Bouzin, C. Dessy, J.-L. Balligand, L. Y. M. Michel

This study compares cardiac remodeling and diastolic dysfunction in C57Bl/6J and C57Bl/6N mice on obesogenic diets, finding strain-specific responses to diet.

bioRxiv · Mar 15, 2024 · PreprintPhysiology

A NRF2/beta3-adrenoreceptor axis drives a sustained antioxidant and metabolic rewiring through the pentose-phosphate pathway to alleviate cardiac stress

L. Michel, H. Esfahani, R. Verdoy, D. de Mulder, J. Ambroise, V. Roelants, B. Bouchard, J. Savary, J. Dewulf, T. Doumont, C. Bouzin, V. Haufroid, J. J. F. P. Luiken, M. Nabben, M. Singleton, , J.-L. Balligand

This study identifies a NRF2/beta3-adrenoreceptor axis that enhances antioxidant defenses and metabolic rewiring via the pentose-phosphate pathway, protecting cardiac function under stress.

bioRxiv · Nov 10, 2023Physiology

Beta-3 Adrenoreceptors protect from hypertrophic remodelling through AMP-Activated Protein Kinase and Autophagy.

E. Dubois-Deruy, R. Gelinas, C. Beauloye, H. Esfahani, C. Dessy, L. Bertrand, J.-L. Balligand

This study shows that beta3-adrenergic receptors reduce cardiac hypertrophy by activating AMPK and preserving autophagy in both in vitro and in vivo models.

bioRxiv · Jan 14, 2020Cell Biology