Author

E. M. Nehls

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:mouse· 1 Organism:non-human primate· 1

Frequent Co-Authors

A. Smirnov· 2 shared C. L. Stallings· 2 shared D. Kreamalmeyer· 2 shared R. L. Kinsella· 2 shared S. K. Naik· 2 shared S. M. Chavez· 2 shared A. Samuels· 1 shared A. Swain· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersMicrobiology

A. Smirnov

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Author profile →

2 papers

Recent·Most cited

Type I IFN signaling mediates NET release to promote Mycobacterium tuberculosis replication and granuloma caseation

C. Sur Chowdhury, R. L. Kinsella, E. M. Nehls, S. K. Naik, D. S. Lane, P. Talukdar, S. M. Chavez, A. Smirnov, W. Beatty, D. Kreamalmeyer, J. T. Mattila, C. L. Stallings

This study shows that type I IFN signaling drives NET release in neutrophils, promoting Mtb replication and granuloma caseation.

bioRxiv · Nov 29, 2022 · PreprintMicrobiology

Autophagy protects against high-dose Mycobacterium tuberculosis infection

S. Feng, E. M. Nehls, R. L. Kinsella, S. M. Chavez, S. K. Naik, S. R. McKee, N. Dubey, A. Samuels, A. Swain, X. Cui, S. V. Hendrix, R. Woodson, D. Kreamalmeyer, A. Smirnov, M. N. Artyomov, , C. L. Stallings

This study shows autophagy in myeloid cells protects against high-dose Mycobacterium tuberculosis infection by limiting myeloid-derived suppressor cells and promoting T cell responses, distinct from its role in low-dose infection.

bioRxiv · Nov 4, 2022Microbiology