Author

E. R. Duke

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:non-human primate· 1 Organism:rat· 1

Frequent Co-Authors

J. T. Schiffer· 3 shared D. B. Reeves· 2 shared E. F. Cardozo-Ojeda· 2 shared A. Goyal· 1 shared A. M. Spivak· 1 shared B. T. Mayer· 1 shared C. W. Peterson· 1 shared H.-P. Kiem· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersMicrobiology

J. T. Schiffer

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3 papers

Recent·Most cited

Mathematical modeling explains differential SARS CoV-2 kinetics in lung and nasal passages in remdesivir treated rhesus macaques

A. Goyal, E. R. Duke, E. F. Cardozo-Ojeda, J. T. Schiffer

The paper develops a mathematical model to explain why remdesivir reduces SARS-CoV-2 viral loads more in lungs than nasal passages in rhesus macaques.

bioRxiv · Jun 21, 2020 · PreprintMicrobiology

Thresholds for post-rebound SHIV control after CCR5 gene-edited autologous hematopoietic cell transplantation

E. F. Cardozo-Ojeda, E. R. Duke, C. W. Peterson, D. B. Reeves, B. T. Mayer, H.-P. Kiem, J. T. Schiffer

This study identifies thresholds for post-rebound SHIV control after CCR5 gene-edited autologous HSPC transplantation using a mathematical model.

bioRxiv · Mar 12, 2020Microbiology

A majority of HIV persistence during antiretroviral therapy is due to infected cell proliferation

D. B. Reeves, E. R. Duke, T. A. Wagner, S. E. Palmer, A. M. Spivak, J. T. Schiffer

The paper uses ecological methods to show that most HIV-infected cells during ART arise from proliferation, not new infections, suggesting proliferation reduction could decrease the viral reservoir.

bioRxiv · Sep 7, 2018Immunology