Author

Elizabeth A. Crinzi

University at Buffalo, State University of New York

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 2

Signature Assays, Organisms & Methods

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Assay:ChIP-seq· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1

Frequent Co-Authors

Emma K. HaleyiD· 2 shared James N. JarvisiD· 2 shared Kaiyu JiangiD· 2 shared Adam Youlin HeiD· 1 shared Charles G. DankoiD· 1 shared Edward Rice· 1 shared Gilad BarshadiD· 1 shared Kerry E. PoppenbergiD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersGenetics

Emma K. HaleyORCID

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2 papers

Recent·Most cited

GeneticsbioRxivPreprintAug 19, 2024

Using Functional Genomic Data in Monocytes/Macrophages and Genotyping to Nominate Disease-Driving Single Nucleotide Polymorphisms and Target Genes in Juvenile Idiopathic Arthritis

Emma K. Haley, Gilad Barshad, Adam Youlin He…·James N. Jarvis

The paper identifies disease-driving SNPs in JIA risk haplotypes using functional genomic data and genotyping, and links them to target genes through chromatin interactions.

1 citation

humanbulk RNA-seq

𝕏 PostBluesky

GeneticsFrontiers in ImmunologyPeer-reviewedSep 29, 2022

Analysis of Chromatin Data Supports a Role For CD14+ Monocytes/Macrophages in Mediating Genetic Risk for Juvenile Idiopathic Arthritis

Elizabeth A. Crinzi, Emma K. Haley, Kerry E. Poppenberg…·James N. Jarvis

The paper uses chromatin data to show JIA risk variants affect innate immunity through enrichment of epigenetic marks in monocytes and macrophages.

9 citations

humanChIP-seq

𝕏 PostBluesky