Author

Emily D. Shaw

University of Michigan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Assay:CRISPR screen· 1 Research Focus:database / resource· 1 Organism:Drosophila· 1

Frequent Co-Authors

Maria do Carmo CostaiD· 3 shared Ana F. FerreiraiD· 2 shared João VasconcelosiD· 2 shared Mafalda RaposoiD· 2 shared Manuela Caroline Ferreira LimaiD· 2 shared Naila S. Ashraf· 2 shared Teresa Taylor KayiD· 2 shared Anna J. Barget· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersNeuroscience

Maria do Carmo CostaORCID

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Author profile →

3 papers

Recent·Most cited

Blood DDIT4 and TRIM13 transcript levels mark the early stages of Machado-Joseph disease

Ana F. Ferreira, Mafalda Raposo, Emily D. Shaw, João Vasconcelos, Teresa Taylor Kay, Conceição Bettencourt, Maria Luiza Saraiva Pereira, Laura Bannach Jardim, Maria do Carmo Costa, Manuela Caroline Ferreira Lima

This study identifies blood transcript levels of DDIT4 and TRIM13 as potential biomarkers for early-stage Machado-Joseph disease, showing high accuracy in distinguishing carriers from controls.

bioRxiv · Oct 1, 2023 · PreprintNeuroscience

Tissue-specific vulnerability to apoptosis in Machado-Joseph disease

Ana F. Ferreira, Mafalda Raposo, Emily D. Shaw, Naila S. Ashraf, Filipa Medeiros, M. Fátima Brilhante, Matthew Perkins, João Vasconcelos, Teresa Taylor Kay, Maria do Carmo Costa, Manuela Caroline Ferreira Lima

The paper assesses apoptosis-related gene expression in MJD patients and mice to identify potential biomarkers and tissue-specific apoptosis vulnerability.

Cells · May 17, 2023Peer-reviewedNeuroscience

Druggable genome screen identifies new regulators of the abundance and toxicity of ATXN3, the Spinocerebellar Ataxia Type 3 disease protein

Naila S. Ashraf, Joanna R. Sutton, Yemen Yang, Bedri Ranxhi, Kozeta Libohova, Emily D. Shaw, Anna J. Barget, Sokol V. Todi, Henry Lauris Paulson, Maria do Carmo Costa

The paper identifies 15 druggable genes that modulate ATXN3 levels and toxicity in SCA3, with FBXL3/SCF pathway showing potential as a therapeutic target.

Neurobiology of Disease · Nov 26, 2019Peer-reviewedNeuroscience