Author

Bedri Ranxhi

Wayne State UniversityORCID 0000-0003-2508-731XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:Drosophila· 3 Approach:experimental assay· 3 Organism:human· 2 Assay:CRISPR screen· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Sokol V. TodiiD· 4 shared Wei‐Ling TsouiD· 3 shared Peter A. LeWittiD· 2 shared Alyson L. SujkowskiiD· 1 shared Anna J. Barget· 1 shared Emily D. Shaw· 1 shared Fatimah H Hussein· 1 shared Henry Lauris PaulsoniD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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4 shared papersNeuroscience

Sokol V. TodiORCID

Mutual co-authors in cluster

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4 papers

Recent·Most cited

NeurosciencebioRxivPreprintJun 16, 2026

Chronic alpha-Synuclein Over-Expression and Ceruloplasmin Challenge Promote Distinct Iron and Redox Responses in M17 Cells

Masoumeh Pourhadi, Bedri Ranxhi, Seth P Sukaria…·Wei‐Ling Tsou

This study investigates how chronic alpha-synuclein over-expression and ceruloplasmin-mediated iron modulation affect iron and redox homeostasis in neuroblastoma cells.

DOI 10.64898/2026.06.15.732494

humanproteomics

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Neurosciencenpj Parkinson s DiseasePeer-reviewedAug 6, 2025

Regulation of polyamine interconversion enzymes affects α-Synuclein levels and toxicity in a Drosophila model of Parkinsons disease

Bedri Ranxhi, Zoya R. Bangash, Zachary M. Chbihi…·Wei‐Ling Tsou

This study shows that manipulating polyamine interconversion enzymes in a Drosophila Parkinson's model affects alpha-synuclein levels and toxicity, suggesting a potential therapeutic target.

12 citationstop 2%

Drosophilaexperimental assay

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NeurosciencebioRxivPreprintNov 7, 2023

Progressive degeneration in a new Drosophila model of Spinocerebellar Ataxia type 7

Alyson L. Sujkowski, Bedri Ranxhi, Matthew V. Prifti…·Wei‐Ling Tsou

This paper develops new Drosophila models of SCA7 with polyQ repeats, showing age- and repeat-length-dependent neurodegeneration and protein aggregation.

DOI 10.1101/2023.11.07.566106

Drosophilaexperimental assay

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NeuroscienceNeurobiology of DiseasePeer-reviewedNov 26, 2019

Druggable genome screen identifies new regulators of the abundance and toxicity of ATXN3, the Spinocerebellar Ataxia Type 3 disease protein

Naila S. Ashraf, Joanna R. Sutton, Yemen Yang…·Maria do Carmo Costa

The paper identifies 15 druggable genes that modulate ATXN3 levels and toxicity in SCA3, with FBXL3/SCF pathway showing potential as a therapeutic target.

22 citations

humanDrosophila

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