Author

Fuying Ma

Emory University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Daniel PuniD· 2 shared Janani ParameswaraniD· 2 shared Jie JiangiD· 2 shared Devesh C. PantiD· 1 shared Jessee Zhang· 1 shared Kedamawit TilahuniD· 1 shared Museer A. LoneiD· 1 shared Myles Dudley· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Daniel PunORCID

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2 papers

Recent·Most cited

Lack of motor defects and ALS-like neuropathology in heterozygous Sptlc1 Exon 2 deletion mice

Devesh C. Pant, Museer A. Lone, Janani Parameswaran, Fuying Ma, Nicole Ziak, Prisha Dutta, Zitong Wang, Daniel Pun, Sumit Verma, Thorsten Hornemann, Jie Jiang

This study creates a mouse model with a heterozygous deletion of Sptlc1 exon 2 and finds no motor defects or ALS-like neuropathology, but homozygous mutants die prematurely.

Life Science Alliance · Jun 20, 2026Peer-reviewedNeuroscience

TMEM106B C-terminal fragments drive nucleocytoplasmic transport failure and TDP-43 mislocalization in the aging human brain

Kedamawit Tilahun, Janani Parameswaran, Myles Dudley, Daniel Pun, Fuying Ma, Jessee Zhang, Tatiana Olivia Bolds, Jie Jiang

This study shows that TMEM106B C-terminal fragments cause nucleocytoplasmic transport failure and TDP-43 mislocalization in aging brains, linking them to neurodegeneration.

bioRxiv · Apr 27, 2026 · PreprintNeuroscience