Author

H. L. Rocha

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Approach:statistical model· 1 Research Focus:trajectory inference· 1

Frequent Co-Authors

C. A. Loomis· 1 shared C. L. Mobini· 1 shared D. M. Gilkes· 1 shared F. Kurtoglu· 1 shared I. Godet· 1 shared J. Metzcar· 1 shared J. N. Weiser· 1 shared K. Konstantinopoulos· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMicrobiology

C. A. Loomis

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Targeting host sialic acids in the upper respiratory tract with a broadly-acting neuraminidase to inhibit influenza virus transmission

M. B. Ortigoza, C. L. Mobini, H. L. Rocha, S. Bartlett, C. A. Loomis, J. N. Weiser

This study demonstrates that blocking both 2,3- and 2,6-sialic acid receptors in infant mice using a broadly-acting neuraminidase inhibits influenza virus transmission.

bioRxiv · Jun 2, 2023 · PreprintMicrobiology

A persistent invasive phenotype in post-hypoxic tumor cells is revealed by novel fate-mapping and computational modeling

H. L. Rocha, I. Godet, F. Kurtoglu, J. Metzcar, K. Konstantinopoulos, S. Bhoyar, D. M. Gilkes, P. Macklin

The paper uses fate-mapping and computational modeling to show that post-hypoxic tumor cells develop an invasive phenotype that promotes tumor expansion into oxygenated regions.

bioRxiv · Dec 30, 2020Cancer Biology