Author

H. Schrezenmeier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Becker· 1 shared A. Bick· 1 shared A. Gilg· 1 shared A. Hoellein· 1 shared A. Knaus· 1 shared A. Link· 1 shared A. Thiel-Bodenstaff· 1 shared B. Bette· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperImmunology

A. Becker

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Author profile →

3 papers

Recent·Most cited

Autoantibodies against Progranulin and IL-1 receptor antagonist due to immunogenic posttranslational isoforms contribute to hyperinflammation in critically ill COVID-19

L. Thurner, N. Fadle, M. Bewarder, I. Kos, E. Regitz, O. Cetin, B. Thurner, Y. Fischer, T. Rixecker, K.-D. Preuss, C. Schormann, F. Neumann, S. Hartmann, T. Bock, D. Kaddu-Mulindwa, , S. Koerper

This study identifies autoantibodies against PGRN and IL-1-Ra in critical COVID-19 patients, linked to hyperphosphorylated isoforms and increased inflammation.

bioRxiv · Apr 23, 2021 · PreprintImmunology

An enzyme-based immunodetection assay to quantify SARS-CoV-2 infection

C. Conzelmann, A. Gilg, R. Gross, D. Schuetz, N. Preising, L. Staendker, B. Jahrsdoerfer, H. Schrezenmeier, K. M. Sparrer, T. Stamminger, S. Stenger, J. Muench, J. A. Mueller

This paper introduces an enzyme-based immunodetection assay to quantify SARS-CoV-2 infection by measuring viral spike protein expression in cell cultures.

Antiviral Research · Sep 1, 2020Peer-reviewedMicrobiology

Complement- and inflammasome-mediated autoinflammation-paroxysmal nocturnal hemoglobinuria

B. Hoechsmann, Y. Murakami, M. Osato, A. Knaus, M. Kawamoto, N. Inoue, T. Hirata, S. Murata, M. Anliker, T. Eggerman, M. Jaeger, R. Floettmann, A. Hoellein, S. Murase, Y. Ueda, , T. Kinoshita

This study identifies AIF-PNH as a disease caused by germline and somatic mutations in PIGT, leading to complement activation and inflammasome-mediated IL-1β secretion, which is treatable with eculizumab.

bioRxiv · May 13, 2019 · PreprintImmunology