Author

I. E. Lundberg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

P.-J. Jakobsson· 2 shared A. Notarnicola· 1 shared A. S. G. Feria· 1 shared B. Horuluoglu· 1 shared C. Fernandes-Cerqueira· 1 shared C. Gronwall· 1 shared C. Hellstrom· 1 shared C. Leijding· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersImmunology

P.-J. Jakobsson

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Author profile →

3 papers

Recent·Most cited

Serum from patients with Idiopathic inflammatory myopathy induces skeletal muscle weakness

C. Leijding, K. M. Andreasson, B. Horuluoglu, A. S. G. Feria, E. Van Gompel, M. Dastmalchi, S. Gastaldello, H. Alexanderson, I. E. Lundberg, D. C. Andersson

This study shows that serum from IIM patients reduces skeletal muscle force, but isolated IgG does not, suggesting humoral factors directly cause muscle weakness.

bioRxiv · Apr 2, 2024Physiology

Autoantigenic properties of the aminoacyl tRNA synthetase family in idiopathic inflammatory myopathies

C. Preger, A. Notarnicola, C. Hellstrom, E. Wigren, C. Fernandes-Cerqueira, H. Idborg, I. E. Lundberg, H. Persson, S. Graslund, P.-J. Jakobsson

The paper identifies novel autoantibodies against aminoacyl tRNA synthetases in idiopathic inflammatory myopathies, expanding the known autoantigenic targets.

bioRxiv · Jun 13, 2022Immunology

Identifying novel B-cell targets for chronic inflammatory autoimmune disease by screening of chemical probes in a patient-derived cell assay

Y. Sundstrom, M.-M. Shang, S. K. Panda, C. Gronwall, F. Wermeling, I. Gunnarsson, I. E. Lundberg, M. Sundstrom, P.-J. Jakobsson, L. Berg

The paper screens chemical probes in a patient-derived cell assay to identify novel B-cell targets for chronic inflammatory autoimmune diseases, finding inhibitors of bromodomain proteins and histone methyl transferases effective in blocking B-cell function.

bioRxiv · Jan 8, 2020Immunology