Author

I. Ghiran

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:electronic health records· 1 Approach:wet-lab / biochemical assay· 1

Frequent Co-Authors

A. Das· 1 shared A. Khamesra· 1 shared A. M. Salvador· 1 shared A. Yeri· 1 shared B. Formosa· 1 shared B. P. Jena· 1 shared C. Xiao· 1 shared D. J. Taatjes· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. Das

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Author profile →

2 papers

Recent·Most cited

Possible Mechanism of Porosome-Mediated Exosome Release

W. J. Cho, B. Formosa, M. G. Zhvania, D. J. Taatjes, I. Ghiran, B. P. Jena

The paper investigates porosome-mediated exosome release by knocking out SPCA1 in Min6 cells, showing reduced glucose-stimulated insulin and exosome release.

bioRxiv · Mar 6, 2022 · PreprintCell Biology

Red blood cell-derived extracellular vesicles mediate intercellular communication in ischemic heart failure

A. Das, N. Valkov, A. M. Salvador, I. Kur, O. Ziegler, A. Yeri, F. C. Garcia, S. Lu, A. Khamesra, C. Xiao, R. Rodosthenous, G. Li, S. Srinivasan, V. Toxavidis, J. Tigges, , S. Das

The paper uses a transgenic mouse model to show RBC-derived extracellular vesicles target cardiomyocytes and microglia in ischemic heart failure, with targeted cells showing gene enrichment in proliferation and metabolism.

bioRxiv · May 2, 2019 · PreprintCell Biology