Author

I. Krueger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Bosbach· 2 shared H. Schelzig· 2 shared K. J. Krott· 2 shared M. Elvers· 2 shared T. Feige· 2 shared A. Beele· 1 shared A. Chadt· 1 shared B. Kehrel· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersPathology

A. Bosbach

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2 papers

Recent·Most cited

Crosstalk between thrombospondin-1 and CD36 modulates platelet-RBC interaction limiting thrombosis and abdominal aneurysm formation

K. J. Krott, T. Feige, A. Bosbach, A. Beele, I. Krueger, F. Reusswig, E. Schickentanz-Dey, A. Chadt, M. Kelm, N. Gerdes, S. Pfeiler, K. Jurk, K. Kiouptsi, C. Reinhardt, H. Al-Hasani, , M. Elvers

This study identifies a novel CD36-TSP-1 signaling pathway that modulates platelet-RBC interaction, promoting thrombus formation and contributing to abdominal aortic aneurysm pathology.

bioRxiv · Mar 18, 2024 · PreprintPathology

Collagen receptor GPVI-mediated platelet activation and pro-coagulant activity aggravates inflammation and aortic wall remodelling in abdominal aortic aneurysm

T. Feige, A. Bosbach, K. J. Krott, J. Mulorz, M. Chatterjee, J. Ortscheid, E. Krueger, I. Krueger, W. Ibing, M. Grandoch, M. U. Wagenhaeuser, H. Schelzig, M. Elvers

This study shows that GPVI-mediated platelet activation worsens AAA by promoting inflammation and aortic wall remodeling, suggesting GPVI as a therapeutic target.

bioRxiv · Nov 20, 2023 · PreprintCell Biology