Author

J. B. Hackett

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

G. Dyson· 2 shared H. M. Gibson· 2 shared J. E. Glassbrook· 2 shared M. Bross· 2 shared M. C. Muniz· 2 shared N. Movahhedin· 2 shared A. Fielder· 1 shared A. Ullrich· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersCancer Biology

G. Dyson

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Author profile →

2 papers

Recent·Most cited

Host genetic background regulates the capacity for anti-tumor antibody-dependent phagocytosis

J. E. Glassbrook, J. B. Hackett, M. C. Muniz, M. Bross, G. Dyson, N. Movahhedin, A. Ullrich, H. M. Gibson

This study shows host genetics influence anti-tumor antibody therapy effectiveness by affecting macrophage phagocytosis capacity in mouse models.

bioRxiv · May 9, 2023 · PreprintCancer Biology

A Diversity Outbred F1 mouse model identifies host-intrinsic genetic regulators of response to immune checkpoint inhibitors

J. B. HACKETT, J. E. Glassbrook, M. C. Muniz, M. Bross, A. Fielder, G. Dyson, J. McCasland, N. Movahhedin, C. E. McCarthy-Leo, H. M. Gibson

The paper identifies prolactin as a host-intrinsic genetic regulator of response to immune checkpoint inhibitors using Diversity Outbred and Collaborative Cross mouse models.

bioRxiv · Oct 7, 2021Immunology