Author

G. Dyson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 5 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1

Frequent Co-Authors

H. M. Gibson· 2 shared J. B. Hackett· 2 shared J. E. Glassbrook· 2 shared M. Bross· 2 shared M. C. Muniz· 2 shared N. Movahhedin· 2 shared A. Aboukameel· 1 shared A. Bollig-Fischer· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersCancer Biology

H. M. Gibson

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

The Hippo kinases control inflammatory Hippo signaling and restrict bacterial infection in eukaryotic phagocytes

B. M. St. Louis, S. M. Quagliato, Y.-T. Su, G. Dyson, P.-C. Lee

This study shows MST1/2 control inflammatory Hippo signaling in macrophages, affecting cytokine release and restricting bacterial infection.

bioRxiv · Dec 8, 2023Microbiology

Host genetic background regulates the capacity for anti-tumor antibody-dependent phagocytosis

J. E. Glassbrook, J. B. Hackett, M. C. Muniz, M. Bross, G. Dyson, N. Movahhedin, A. Ullrich, H. M. Gibson

This study shows host genetics influence anti-tumor antibody therapy effectiveness by affecting macrophage phagocytosis capacity in mouse models.

bioRxiv · May 9, 2023 · PreprintCancer Biology

Hyperglycemic O-GlcNAc transferase activity drives cancer stem cell induction in TNBC

S. Ayodeji, B. Bao, E. A. Teslow, L. A. Polin, G. Dyson, A. Bollig-Fischer, C. Fehl

This study identifies a hyperglycemia-driven OGT-TET1-TARDBP pathway that promotes cancer stem cell induction in TNBC, linking obesity to increased TNBC risk.

bioRxiv · Mar 14, 2022Cancer Biology

Molecular profiling of XPO1 inhibitor and gemcitabine-nab-paclitaxel combination in cellular and LSL-Kras G12D/+; Trp53 fl/+; Pdx1-Cre (KPC) pancreatic cancer model

M. H. Uddin, A. Aboukameel, Y. Li, H. Y. Khan, R. E. Sexton, S. Bannoura, G. Dyson, M. N. Al-Hallak, Y. Mzannar, A. Mohamed, Y. Landesman, S. Kim, R. Beydoun, R. M. Mohammad, A. F. Shields, A. S. Azmi

This study investigates the synergy between XPO1 inhibitor selinexor and gemcitabine-nab-paclitaxel in pancreatic cancer using cellular and mouse models, showing improved survival and molecular changes.

bioRxiv · Mar 8, 2022 · PreprintCancer Biology

A Diversity Outbred F1 mouse model identifies host-intrinsic genetic regulators of response to immune checkpoint inhibitors

J. B. HACKETT, J. E. Glassbrook, M. C. Muniz, M. Bross, A. Fielder, G. Dyson, J. McCasland, N. Movahhedin, C. E. McCarthy-Leo, H. M. Gibson

The paper identifies prolactin as a host-intrinsic genetic regulator of response to immune checkpoint inhibitors using Diversity Outbred and Collaborative Cross mouse models.

bioRxiv · Oct 7, 2021Immunology