Author

J. B. Rose

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

H. Chen· 2 shared A. Hjelmeland· 1 shared A. M. Carter· 1 shared B. Herring· 1 shared B. Ren· 1 shared C. Graham· 1 shared C. Tan· 1 shared E. A. Woltering· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

H. Chen

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2 papers

Recent·Most cited

PKD-1 Signaling Is Required for the Maintenance of CSCs with Epithelial-mesenchymal Plasticity in Pancreatic Neuroendocrine Tumors

Y. Guo, Y. Jiang, J. B. Rose, R. Jaskula-Sztul, A. Hjelmeland, H. Chen, B. Ren

This study shows PKD-1 signaling is essential for maintaining CSCs with epithelial-mesenchymal plasticity in pNETs, suggesting it as a therapeutic target.

bioRxiv · Feb 17, 2022 · PreprintCancer Biology

Cdk5 drives formation of heterogeneous pancreatic neuroendocrine tumors

A. M. Carter, N. Kumar, B. Herring, C. Tan, R. Guenter, R. Telange, W. Howse, F. Viol, C. Graham, T. R. McCaw, h. Bickerton, F. Gillardon, E. A. Woltering, J. Totenhagen, R. Banerjee, , J. Bibb

This paper develops a mouse model of PanNETs driven by Cdk5 and shows its relevance to human tumors, supporting Cdk5 as a therapeutic target.

bioRxiv · May 25, 2021Cancer Biology