Author

J. J. Verschuuren

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

M. G. Huijbers· 2 shared S. M. van der Maarel· 2 shared Y. E. Fillie-Grijpma· 2 shared A. J. van der Sluijs-Gelling· 1 shared C. Gstottner· 1 shared D. L. E. Vergoossen· 1 shared E. Dominguez-Vega· 1 shared J. J. M. van Dongen· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

M. G. Huijbers

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2 papers

Recent·Most cited

Autoantibody subclass predominance is not driven by aberrant class switching or impaired B cell development

L. M. Paardekooper, Y. E. Fillie-Grijpma, A. J. van der Sluijs-Gelling, M. Zlei, R. van Doorn, M. H. Vermeer, M. Paunovic, M. J. Titulaer, S. M. van der Maarel, J. J. M. van Dongen, J. J. Verschuuren, M. G. Huijbers

The paper investigates why IgG4 autoantibodies predominate in certain autoimmune diseases and finds that B cell development is normal, suggesting antigen-driven subclass predominance rather than B cell dysfunction.

bioRxiv · Jun 30, 2023Immunology

Functional monovalency amplifies the pathogenicity of anti-MuSK IgG4 in myasthenia gravis

D. L. E. Vergoossen, J. J. Plomp, C. Gstottner, Y. E. Fillie-Grijpma, R. Augustinus, R. Verpalen, M. Wuhrer, P. W. H. I. Parren, E. Dominguez-Vega, S. M. van der Maarel, J. J. Verschuuren, M. G. Huijbers

This study shows that monovalent anti-MuSK IgG4 antibodies enhance pathogenicity in myasthenia gravis compared to their bivalent form.

bioRxiv · Sep 24, 2020Immunology