Author

J. K. Duncan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:host-pathogen & immune response· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:microscopy & imaging analysis· 1 Assay:proteomics· 1

Frequent Co-Authors

B. C. Russo· 3 shared M. B. Goldberg· 3 shared A. L. Wiscovitch· 2 shared A. C. Hachey· 1 shared

Activity by Year

2019-2019 · peak 3/yr

Co-Author Collaboration Network

4 top collaborators · 6 mutual co-author links

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3 shared papersMicrobiology

B. C. Russo

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3 papers

Recent·Most cited

Shigella flexneri disruption of host cell-cell tension promotes intercellular spread

J. K. Duncan, A. L. Wiscovitch, M. B. Goldberg, B. C. Russo

This study shows that Shigella flexneri uses IpaC to reduce cell-cell tension, enabling intercellular spread by forming protrusions.

bioRxiv · Oct 2, 2019Microbiology

Activation of Shigella flexneri type 3 secretion requires a host-induced conformational change to the translocon pore

B. C. Russo, J. K. Duncan, A. L. Wiscovitch, A. C. Hachey, M. B. Goldberg

This paper shows that host intermediate filaments induce conformational changes in the Shigella T3SS pore, enabling docking and effector secretion.

bioRxiv · Jun 19, 2019Microbiology

Topological analysis of the type 3 secretion system translocon pore protein IpaC following its native delivery to the plasma membrane during infection

B. C. Russo, J. K. Duncan, M. B. Goldberg

The paper determines the topology of IpaC and SipC translocon pore proteins in their native context during infection, resolving conflicting data and identifying topological differences that may underlie functional divergence.

bioRxiv · Apr 11, 2019Microbiology