J. Martinez-Pinna

G protein-coupled estrogen receptor activation by Bisphenol-A disrupts protection from apoptosis conferred by estrogen receptors ERα and ERβ in pancreatic beta cells

This paper shows that BPA induces apoptosis in pancreatic beta cells by reducing ERβ heterodimers, while GPER activation by G1 has a similar effect, highlighting a novel mechanism of endocrine disruption.

Bisphenol-S and Bisphenol-F alter mouse pancreatic β-cell ion channel expression and activity and insulin release through an estrogen receptor ERβ mediated pathway

This study shows BPS and BPF affect mouse β-cell ion channels and insulin release via ERβ pathways, with effects absent in ERβ knockout mice.