R. M. Medina-Gali

The paper tests six chemicals on human and rodent β-cells to identify metabolism-disrupting chemicals with diabetogenic activity, finding PFOA reduces glucose-stimulated insulin secretion.

This paper shows that BPA induces apoptosis in pancreatic beta cells by reducing ERβ heterodimers, while GPER activation by G1 has a similar effect, highlighting a novel mechanism of endocrine disruption.

This study shows BPS and BPF affect mouse β-cell ion channels and insulin release via ERβ pathways, with effects absent in ERβ knockout mice.