Author

James Woods

New York Stem Cell FoundationPrimary ORCID profile (0000-0002-9171-0723 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

James WoodsiD1 paper · Broad Institute

Biology Disciplines

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Approach:genetic perturbation / CRISPR· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Bjarki JóhannessoniD· 1 shared Deepta BhattacharyaiD· 1 shared Eric O. LongiD· 1 shared Fei Mo· 1 shared Frederick J. MonsmaiD· 1 shared Hannah A. PizzatoiD· 1 shared Henning UrlaubiD· 1 shared John P. AtkinsoniD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

Bjarki JóhannessonORCID

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2 papers

Recent·Most cited

ImmunologyStem Cell ReportsPeer-reviewedJan 11, 2024

Engineering Human Pluripotent Stem Cell Lines to Evade Xenogeneic Transplantation Barriers

Hannah A. Pizzato, Paula Alonso‐Guallart, James Woods…·Deepta Bhattacharya

This study genetically modifies human pluripotent stem cells to evade immune rejection in mice by knocking out HLA genes and adding inhibitory factors, leading to persistent teratomas in immunocompetent models.

28 citations

mousegenetic perturbation / CRISPR

𝕏 PostBluesky

ImmunologybioRxivPreprintApr 3, 2023

Functional genomics identifies extension of complex N-glycans as a mechanism to evade lysis by natural killer cells

Xiaoxuan Zhuang, James Woods, Yanlong Ji…·Eric O. Long

The paper identifies SPPL3 as a gene that enables tumor cells to evade NK cell lysis by altering N-glycan structures, and shows that inhibiting N-glycan maturation restores NK sensitivity.

DOI 10.1101/2023.04.03.535404

humanproteomics

𝕏 PostBluesky