Author

Jasmine Wong

Agency for Science, Technology and Research

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Genetic & Genomic Medicine· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:GWAS / association mapping· 1 Organism:mouse· 1

Frequent Co-Authors

Akiko ShimamuraiD· 1 shared Annick Raas‐RothschildiD· 1 shared Benjamin L. FarahiD· 1 shared Bertrand BoissoniD· 1 shared Bruno ReversadeiD· 1 shared Cassandra R. HarapasiD· 1 shared Chien‐Hsiung YuiD· 1 shared Devon E. BonneriD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperGenetic & Genomic Medicine

Akiko ShimamuraORCID

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2 papers

Recent·Most cited

A Cross-Species Systems Genetics Framework Identifies Causal Genes in Diabetic Nephropathy

Kunal Mishra, Rashidah Binte Sakban, Sujithra Shankar, Jasmine Wong, Benjamin L. Farah, Jing Guo, Jianhong Ching, Ming Shen Tham, Jean‐Paul Kovalik, Susan B. Gurley, Enrico G. Petretto, Nicholas S. Tolwinski, Thomas M. Coffman, Jacques V. Behmoaras

This paper identifies causal genes in diabetic nephropathy using a cross-species systems genetics framework, finding 192 candidate genes with 40% validated in humans.

bioRxiv · Jul 1, 2026 · PreprintSystems Biology

DPP9 deficiency: an inflammasomopathy which can be rescued by lowering NLRP1/IL-1 signaling

Cassandra R. Harapas, Kim S. Robinson, Kenneth Lay, Jasmine Wong, Ricardo Moreno Traspas, Nasrin Nabavizadeh, Annick Raas‐Rothschild, Bertrand Boisson, Scott B. Drutman, Pawat Laohamonthonkul, Devon E. Bonner, Jingwei Rachel Xiong, Mark D. Gorrell, Sophia Davidson, Chien‐Hsiung Yu, , Bruno Reversade

This study identifies DPP9 deficiency as a Mendelian disorder causing inflammasome hyperactivity, which can be rescued by reducing NLRP1/IL-1 signaling in mice.

Science Immunology · Sep 16, 2022Peer-reviewedGenetic & Genomic Medicine