Author

Kim S. Robinson

University of YorkORCID 0000-0003-1182-8789Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Franklin L. ZhongiD· 2 shared Akiko ShimamuraiD· 1 shared Alexander J. Hogg· 1 shared Alison LaytoniD· 1 shared Annick Raas‐RothschildiD· 1 shared Arnel Junior Villamin· 1 shared Bertrand BoissoniD· 1 shared Bruno ReversadeiD· 1 shared

Activity by Year

2022-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersImmunology

Franklin L. ZhongORCID

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintOct 20, 2025

Non-canonical inflammasome drives intrinsic anti-microbial responses in human Natural Killer (NK) cells.

Nathan Georges François Leborgne, Jadie Acklam, Alexander J. Hogg…·Dave Boucher

This study identifies a non-canonical inflammasome in human NK cells that mediates anti-microbial responses against Salmonella without canonical inflammasome components.

DOI 10.1101/2025.10.20.682800

humanproteomics

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ImmunologybioRxivPreprintAug 13, 2025

Clarifying the priming requirement and activator specificity for the NLRP3 inflammasome in human keratinocytes in vitro

Pritisha Rozario, Ying Shiang Lim, Shirley Suet Lee Ding…·Franklin L. Zhong

This study investigates NLRP3 inflammasome activation in human keratinocytes, showing it is induced by IFN-gamma but not LPS, and that specific activators like Hla target NLRP3 while nigericin activates both NLRP1 and NLRP3.

3 citations

humanexperimental assay

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Genetic & Genomic MedicineScience ImmunologyPeer-reviewedSep 16, 2022

DPP9 deficiency: an inflammasomopathy which can be rescued by lowering NLRP1/IL-1 signaling

Cassandra R. Harapas, Kim S. Robinson, Kenneth Lay…·Bruno Reversade

This study identifies DPP9 deficiency as a Mendelian disorder causing inflammasome hyperactivity, which can be rescued by reducing NLRP1/IL-1 signaling in mice.

58 citationstop 5%

humanstatistical model

𝕏 PostBluesky