Author

Jiankun Zhu

The University of Texas Southwestern Medical CenterPrimary ORCID profile (0000-0002-2424-6692 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Jiankun ZhuiD1 paper · The University of Texas Southwestern Medical Center

Biology Disciplines

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Developmental Biology· 1

Signature Assays, Organisms & Methods

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Assay:ATAC-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Beverly A. RothermeliD· 1 shared Brian F. NiemeyeriD· 1 shared Edwin Rosado-Olivieri· 1 shared Guilherme Henrique Souza BomfimiD· 1 shared Joaquin Maximiliano EspinosaiD· 1 shared Malay ChakladeriD· 1 shared Neil JejuiD· 1 shared Rodrigo S. LacruziD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperDevelopmental Biology

Beverly A. RothermelORCID

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1 paper

Recent·Most cited

Elevated mitochondrial metabolism in Down syndrome iPSCs reduces commitment to neuroectoderm

Malay Chaklader, Guilherme Henrique Souza Bomfim, Jiankun Zhu, Neil Jeju, Théodore Nguyen, Yingli Duan, Brian F. Niemeyer, Joaquin Maximiliano Espinosa, Edwin Rosado-Olivieri, Weichun Lin, Rodrigo S. Lacruz, Beverly A. Rothermel

This study shows that increased mitochondrial metabolism in Down syndrome iPSCs impairs neuroectoderm commitment by reducing ciliation, and this effect can be reversed by inhibiting mitochondrial calcium uptake.

EMBO Reports · Oct 30, 2026Peer-reviewedDevelopmental Biology