Author

Julia Dallmann

Max Planck Institute for Heart and Lung Research

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Assay:spatial transcriptomics· 1 Organism:zebrafish· 1

Frequent Co-Authors

Arica BeisawiD· 1 shared Bai‐Lin WuiD· 1 shared Didier Y. R. StainieriD· 1 shared Florian ConstantyiD· 1 shared I-Ting LiniD· 1 shared Ke-Hsuan WeiiD· 1 shared Rashmi PriyaiD· 1 shared Shih-Lei LaiiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

Arica BeisawORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Border-zone cardiomyocytes and macrophages contribute to remodeling of the extracellular matrix to promote cardiomyocyte invasion during zebrafish cardiac regeneration

Florian Constanty, Bai‐Lin Wu, Ke-Hsuan Wei, I-Ting Lin, Julia Dallmann, Stefan Günther, Till Lautenschlaeger, Rashmi Priya, Shih-Lei Lai, Didier Y. R. Stainier, Arica Beisaw

This study identifies border-zone cardiomyocytes and macrophages as key contributors to ECM remodeling that facilitates cardiomyocyte invasion during zebrafish cardiac regeneration.

Nature Communications · Apr 23, 2025Peer-reviewedCell Biology