Author

Julien Menendez

University of California, Santa Cruz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Lindsay HinckiD· 2 shared Rut MolinuevoiD· 2 shared Adele XuiD· 1 shared Davide RuggeroiD· 1 shared Elisa FazzariiD· 1 shared Gwen ThomasiD· 1 shared Harrison J. TomiD· 1 shared Hunter E. SchweigeriD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

Lindsay HinckORCID

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2 papers

Recent·Most cited

Defects in the DNA Damage Response of Patient-derived Endometriosis Stromal Cells

Kora Cadle, Gwen Thomas, Hunter E. Schweiger, Julien Menendez, Rut Molinuevo, Lindsay Hinck

This study shows that endometriosis patient-derived stromal cells have a defective DNA damage response, leading to reduced senescence and altered DDR activation under replication stress.

bioRxiv · Oct 29, 2025 · PreprintCell Biology

Subfunctionalized expression drives evolutionary retention of ribosomal protein paralogs in vertebrates

Adele Xu, Rut Molinuevo, Elisa Fazzari, Harrison J. Tom, Zijian Zhang, Julien Menendez, Kerriann M. Casey, Davide Ruggero, Lindsay Hinck, Jonathan K. Pritchard, Maria Barna

The paper investigates the evolutionary retention of ribosomal protein paralogs eS27 and eS27L by showing their subfunctionalized expression patterns are necessary for maintaining total protein expression across cell types.

eLife · Jun 12, 2023Peer-reviewedGenetics