Author

K. A. Cottrell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:proteomics· 1

Frequent Co-Authors

J. D. Weber· 3 shared C.-P. Kung· 1 shared E. A. Bross· 1 shared E. R. Bramel· 1 shared L. Maggi· 1 shared L. S. Torres· 1 shared R. C. Chiou· 1 shared R. D. Kladney· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

9 top collaborators · 23 mutual co-author links

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3 shared papersCancer Biology

J. D. Weber

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Author profile →

3 papers

Recent·Most cited

8-azaadenosine and 8-chloroadenosine are not selective inhibitors of ADAR

K. A. Cottrell, L. S. Torres, J. D. Weber

This paper shows that 8-azaadenosine and 8-chloroadenosine are not selective ADAR inhibitors, as they affect ADAR-dependent and independent cell lines equally and do not activate PKR or alter A-to-I editing.

bioRxiv · May 12, 2021Cancer Biology

ARF suppresses 5'-terminal oligopyrimidine mRNA translation

K. A. Cottrell, R. C. Chiou, J. D. Weber

The paper shows ARF suppresses 5'-terminal oligopyrimidine mRNA translation and identifies related regulators in cancer cells.

bioRxiv · Feb 7, 2020Cell Biology

ADAR1 editing dependency in triple-negative breast cancer

C.-P. Kung, K. A. Cottrell, S. Ryu, E. R. Bramel, R. D. Kladney, E. A. Bross, L. Maggi, J. D. Weber

This study shows ADAR1 is essential for TNBC cell proliferation and tumorigenesis, with its loss reducing proliferation and being rescued by IFNAR1 reduction.

bioRxiv · Jan 31, 2020Cancer Biology