Author

K. Cunatova

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 2 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:metabolism & bioenergetics· 1 Assay:proteomics· 1

Frequent Co-Authors

E. Fernandez-Vizarra· 2 shared J. Houstek· 2 shared M. Vrbacky· 2 shared M. Zeviani· 2 shared P. Pecina· 2 shared T. Mracek· 2 shared A. Cabrera-Orefice· 1 shared A. Pecinova· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBiochemistry

E. Fernandez-Vizarra

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

COA8 is a COX10-binding protein involved in the early biogenesis of cytochrome c oxidase

M. Brischigliaro, K. Cunatova, A. Cabrera-Orefice, J. Pei, C. Franchin, M. Roverso, S. Povea-Cabello, S. Bogialli, G. Arrigoni, S. Arnold, Q. Cong, M. Zeviani, C. Viscomi, E. Fernandez-Vizarra

This study identifies COA8 as a COX10-binding protein essential for COX10 stability and catalytic activity in human cytochrome c oxidase biogenesis.

bioRxiv · Apr 2, 2024 · PreprintBiochemistry

Physiological variability in mitochondrial rRNA predisposes to metabolic syndrome

P. Pecina, K. Cunatova, V. Kaplanova, G. Puertas-Frias, J. Silhavy, K. Tauchmannova, M. Vrbacky, T. Cajka, O. Gahura, M. Pravenec, J. Houstek, T. Mracek, A. Pecinova

The paper investigates how mitochondrial rRNA sequence variation contributes to insulin resistance and metabolic syndrome in rats fed a high-fat diet.

bioRxiv · Nov 8, 2023Physiology

Loss of COX4i1 leads to combined respiratory chain deficiency and impaired mitochondrial proteosynthesis

K. Cunatova, D. Pajuelo Reguera, M. Vrbacky, E. Fernandez-Vizarra, S. Ding, I. Fearnley, M. Zeviani, J. Houstek, T. Mracek, P. Pecina

This study shows that loss of COX4i1 causes combined respiratory chain deficiency and impaired mitochondrial proteosynthesis by disrupting cI and cIV biogenesis.

bioRxiv · Jan 31, 2020 · PreprintBiochemistry