Author

K. Evangelou

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:host-pathogen & immune response· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:imaging· 1

Frequent Co-Authors

A. G. Tzioufas· 2 shared A. Polyzou· 2 shared C. Kittas· 2 shared D. Veroutis· 2 shared N. Lagopati· 2 shared S. Havaki· 2 shared V. G. Gorgoulis· 2 shared A. Angelopoulou· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCell Biology

A. G. Tzioufas

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Senescent cells in Giant Cell Arteritis have inflammatory phenotype participating in tissue injury via IL-6 dependent pathways

D. Veroutis, O. D. Argyropoulou, A. V. Goules, K. Kambas, D. A. Palamidas, K. Evangelou, S. Havaki, A. Polyzou, E. Xingi, E. Karatza, B. A. Kyriaki, A. Cavazza, C. Kittas, D. Thanos, C. Ricordi, , A. G. Tzioufas

This study identifies senescent cells in Giant Cell Arteritis (GCA) arteries with an inflammatory phenotype linked to IL-6 dependent pathways contributing to tissue injury.

bioRxiv · May 19, 2023 · PreprintCell Biology

SARS-CoV-2 infects lung epithelial cells and induces senescence and an inflammatory response in patients with severe COVID-19

K. Evangelou, D. Veroutis, P. G. Foukas, K. Paschalaki, N. Lagopati, M. Dimitriou, A. Papaspyropoulos, O. Hazapis, A. Polyzou, S. Havaki, A. Kotsinas, C. Kittas, A. G. Tzioufas, L. de Leval, D. Vassilakos, , V. Gorgoulis

This study shows SARS-CoV-2 infects alveolar type-2 cells in severe COVID-19 patients, inducing senescence and proinflammatory cytokine expression.

bioRxiv · Jan 2, 2021 · PreprintCell Biology

Genomic instability is an early event driving chromatin reorganization and escape from oncogene-induced senescence

C. Zampetidis, P. Galanos, A. Angelopoulou, Y. Zhu, T. Karamitros, N. Lagopati, R. Mirzazadeh, A. Polyzos, S. Garnerone, E. G. Gusmao, K. Sofiadis, D. E. Pefani, M. Demaria, N. Crosetto, A. Maya-Mendoza, , V. G. Gorgoulis

The paper identifies an early chromosomal inversion that enables escape from oncogene-induced senescence by activating BHLHE40 and promoting malignant transformation.

bioRxiv · Dec 20, 2020Cell Biology